Superoxide dismutase functional regulation in neonatal hypoxia

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Superoxide dismutase functional regulation in neonatal hypoxia

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dc.contributor.author Paulose, C S
dc.contributor.author Athira, Babu
dc.contributor.author Anju, T R
dc.date.accessioned 2010-03-30T06:56:51Z
dc.date.available 2010-03-30T06:56:51Z
dc.date.issued 2009-04
dc.identifier.citation Cochin University of Science and Technology en_US
dc.identifier.other Indian Journal of Biochemistry and Biophysics, Vol. 46, April 2009,pp. 166-171
dc.identifier.uri http://dyuthi.cusat.ac.in/purl/1620
dc.description.abstract Hypoxia is one of the major causes of damage to the fetal and neonatal brain and cardiac functions. in earlier studies we have reported the brain damage caused by hypoxia and resusciation with oxygen and epinephrine and have found that glucose treatment to hypoxic rats and hypoxic rats treated with oxygen shows a reversal of brain damage. during this study the findings may have clinical significance in the proper management of heart and brain functions. en_US
dc.language.iso en en_US
dc.publisher Indian Journal of Biochemistry and Biophysics en_US
dc.subject Super oxide dismutase en_US
dc.subject Neonatal hypoxia en_US
dc.subject Free radical en_US
dc.subject Catalase en_US
dc.subject Glucose en_US
dc.subject Oxygen en_US
dc.subject Epinephrine en_US
dc.title Superoxide dismutase functional regulation in neonatal hypoxia en_US
dc.title.alternative Effect of glucose, oxygen and epinephrine en_US
dc.type Working Paper en_US


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